We build research platforms around endogenous lipid signaling, designing systems that support motivation, routine, and sustained training behavior.
Beyond selling supplements, we study how lipid-mediated signaling systems interact with physical activity. Our work focuses on designing environments where exercise itself becomes the trigger for motivation.
We focus on lipid mediators that interact with exercise-induced signaling, including pathways associated with blood–brain barrier accessibility.
Rather than mimicking stimulation, our research explores how physical activity itself activates endogenous systems related to motivation, engagement, and reward.
Motivation is not added.
It is unlocked by movement.
N-acylethanolamide biology, translated into structured performance systems.
The NAE family is involved in lipid-mediated signaling related to appetite, inflammation, and neural activity. Our platform studies how these pathways contribute to routine, consistency, and exercise-driven motivation—not short-term stimulation.
Peripheral-first lipid signaling for daily training consistency.
This platform focuses on NAEs primarily associated with peripheral signaling, supporting appetite rhythm, physical comfort, and baseline readiness. Designed for stability, repeatability, and long-term routine support.
Exercise-activated lipid signaling beyond the periphery.
We explore NAEs associated with central nervous system engagement, focusing on motivation, reward, and exercise-associated affect. This work remains research-driven and exploratory.
Supplementation is only one layer.
We study how ritual, routine, and feedback loops interact with lipid signaling and physical activity, designing systems that support long-term training adherence.
N-acylethanolamides (NAEs) are endogenous lipid signaling molecules naturally produced in the human body. They participate in peripheral and central pathways related to energy balance, inflammatory signaling, and neural engagement.
Many NAE-related pathways become most relevant during and after physical activity. We study NAEs in the context of movement-triggered signaling, not passive consumption.
Ongoing findings are published separately in Research Updates.
Research results, progress, experimental data → separate "Research Updates" section
Scope: Daily baseline · Routine stability · Peripheral signaling
This platform integrates OEA, PEA, and SEA as a system, not isolated ingredients. Each component contributes to a different peripheral context, together supporting the conditions required for consistent training behavior.
This is not a stimulant stack.
It is a peripheral signaling environment.
Scope: Motivation · Reward · Cognitive engagement
This research track focuses on DHEA (Synaptamide) and AEA (Anandamide)—lipid mediators associated with blood–brain barrier accessibility and exercise-related neural signaling.
Rather than targeting these pathways directly, we study how physical activity itself acts as the primary trigger for endogenous motivation-related signaling.
Central NAE research is conducted with strict boundaries, emphasizing safety, evidence, and biological context.
Identity-verified, impurity-aware, traceable by design.
Lipid systems demand oxidation-aware formulation and packaging.
Repeatable processes over spectacle.
